Multidisciplinary Cardiovascular Annals

Research Article

01/Jan/2009

Surgical Treatment of Chronic Constrictive Pericarditis, Is Tuberculosis Still a Common Cause?

Alireza Alizadeh Ghavidel, Hossein Javadpour

Research Article

01/Jan/2009

Early post operative assessment of CABG by multi-detector row computed tomography and correlation between calcium score and early graft occlusion after coronary bypass surgery

s Rafieian

Research Article

01/Jan/2009

Endoscopic Vein Harvesting in CABG Surgery an Initial Iranian Experience

M Ladan, Daryoosh Javidi

the minimally invasive endoscopic dissection of vessel conduits is steadily gaining acceptance as a preferable alternative to the standard open-incision technique. . As experience and refinements in instrumentation progress, the endoscopic approach will undoubtedly become the procedure of choice for harvesting vessel conduits. This article provides a practical primer, based on our serial experience with endoscopic vein dissections, for those considering the minimally invasive endoscopic approach inharvesting vessels for CABG

Review Article

10/Sep/2016

Pathophysiology of lactic acidosis, and its clinical importance after cardiac surgery

Daryoosh Javidi

In 1925, Clausen identified the accumulation of lactic acid in blood as a cause of acid-base disorder. Several decades later, Huckabee's seminal work firmly established that lactic acidosis frequently accompanies severe illnesses and that tissue hypoperfusion underlies the pathogenesis. In their classic 1976 monograph, Cohen and Woods classified the causes of lactic acidosis according to the presence or absence of adequate tissue oxygenation.The normal blood lactate concentration in unstressed patients is 0.5-1 mmol/L. Patients with critical illness can be considered to have normal lactate concentrations of less than 2 mmol/L. Hyperlactatemia is defined as a mild-to-moderate persistent increase in blood lactate concentration (2-5 mmol/L) without metabolic acidosis, whereas lactic acidosis is characterized by persistently increased blood lactate levels (usually >5 mmol/L) in association with metabolic acidosis (pH < 7.35).1,2 Lactic acidosis is associated with major metabolic dysregulation, tissue hypoperfusion, effects of certain drugs or toxins, or congenital abnormalities in carbohydrate metabolism. Cohen and Woods divided lactic acidosis into 2 categories: “type A”, associated with impaired delivery of oxygen to tissues (DO2) eg, hypotension, cyanosis, cool and clammy extremities, and “type B”, where lactic acidosis occurs in the presence of normal DO2

Review Article

08/Jan/2009

A Peripatetic Discourse on Sildenafil

G Jami Shakibi

Far from being a muni or a yogi, many years have passed since I have lost all the desire to write medical articles for medical journals. Along with this several other desires have also died in me. The following is a peripatetic discourse, not a formal paper, on sildenafil, in response to a request repeated several times by a close Friend of Old Times, Dr M.A.Youssefnia, now a prominent cardiac surgeon at several hospitals, president of the Cardiac Surgeon’s Society, and many other professional organizations.
The imitation of the occidental methods, including “publish or perish” motto, has driven the underdeveloped world to the brink of insanity and chaos, with a mushrooming of many sham journals, seminars, conventions, workshops etc, etc most of which are ruminations of works already published, or mock imitation of original research works, with a premeditated, selective “publish or perish “effect at local levels

Case Report

01/Jan/2009

Takayasu disease and cardiac manifestations, presentation of a case with Takayasu and coronary artery disease

Ali Sadeghpour Tabaee

Case Report

01/Jan/2009

Free floating clot in inferior vena cava with extension to Right Heart: What is the best treatment?

M.H. Ghafarinejad, shahreyar Mali

Multidisciplinary Cardiovascular Annals: Jan 2009, 2 (3), 7 articles.